Rispen is a second-generation antipsychotic that has an affinity for D2, 5-HT2A, alpha 1, alpha 2 and H1 receptors. The mechanism of action of Rispen is not fully understood, current theories focus mainly on its ability to block D2 and 5-HT2A receptors. Rispen was synthesized based on attempts to replicate clozapine effectiveness without its side effects profile. The goal was to develop a drug with low risk of extrapyramidal symptoms (EPS) based on the assumption that a high 5-HT2A/D2 ratio could confer this property [3]. However, when prescribed at higher doses, Rispen produces EPS consistently [4], indicating that 5-HT2A antagonism alone cannot eliminate EPS associated with substantial D2 receptor blockade.
Other names for this medication:
Risperidone,
Risperdal,
Adovia,
Belivon,
Denoral,
Depolan,
Muistin,
Nodiril,
Perdamel,
Prospera,
Radigen,
Rispen,
Risper,
Risperidona,
Rispolux,
Rispond,
Rispone,
Sequinan,
Sizodon,
Torendo,
Zargus,
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Depramil,
Aurex,
Citol,
Tromic,
Gloclav,
Ishape,
Ulcesep,
Zenegra,
Zanprol,
Calutide,
Rofex,
Zepatier,
Sievert,
Socalm,
Stronazon,
Propump,
Luvox,
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Trazodon,
Gabanet,
Epinor,
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